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Metabolic enzyme PFKFB4 activates transcriptional coactivator SRC-3 to drive breast cancer

doi: 10.1056/NEJM198712313172706 10 MedinaMQuesadaA

Encapsulation and scaffold are based on pharmaceutical science that isolates islets from various immune cells and provides them with an appropriate microenvironment

In addition, the oldest orphan nuclear receptor estrogen-related receptor (ERR), an important regulator of metabolic gene transcription and innate immune function, including those induced by toll-like receptor (TLR) and antimicrobial activities against intracellular bacterial infection [147], facilitated post-translational activation of autophagy by deacetylating various autophagy-related proteins, including ATG5, BECN1, and ATG7