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Firstly, by dampening inflammatory responses integral to the pathophysiology of depression and anxiety, omega-3 PUFAs and their metabolites can inhibit leukocyte chemotaxis, reduce the expression of adhesion molecules, decrease the production of pro-inflammatory mediators, and encourage a shift in microglia polarization towards a reparative, non-inflammatory state (75, 76)

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Research has shown that targeting TET2 can disrupt the immunosuppressive activity of MDSCs and TAMs, thereby enhancing the efficacy of adaptive cellular therapies [99]

1 2 3 Lithium in Neuropsychiatry: The Comprehensive Guide