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Korde AS, Pettigrew LC, Craddock SD, Maragos WF (2005) The mitochondrial uncoupler 2,4-dinitrophenol attenuates tissue damage and improves mitochondrial homeostasis following transient focal cerebral ischemia

Studies have shown that in various neurodegenerative conditions, DAAs or reactive astrocytes exhibit increased glycolytic activity alongside impaired mitochondrial oxidative phosphorylation (Jiang and Cadenas, 2014

The latter strains may have the VacA m2 gene without the CagPAI, which could make H

A disruption in calcium homeostasis leads to the formation of A plaques, the accumulation of neurofibrillary tangles, and the dysfunction of synaptic plasticity, leading to delayed neurodegenerative changes in the brain, excitotoxicity, and glial cell-induced neuroinflammation (Chanaday et al., 2021)