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This results in preventing bile acid reabsorption thus decreasing the bile acid pool, which in turn reduces FGF15/19 and increases the gene transcription of the bile acid synthesis rate-limiting enzyme, CY7A1

In the pivotal, phase 3, placebo-controlled RESPONSE trial (NCT04620733), seladelpar, a novel delpar (selective PPAR-delta agonist), led to a significantly higher percentage of PBC patients achieving the composite biochemical endpoint (ALP 0 U/L from baseline) and safety by baseline ALP subgroup were also evaluated