glutathione ferroptosis gpx4 methylation puts a brake on Oncology Letters

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Bernuzzi, F., Recalcati, S., Alberghini, A

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GSH is essential for immunomodulation of both innate and adaptive immune system functions, including T-lymphocyte proliferation, polymorphonuclear neutrophil phagocytosis, and dendritic cell functions, and is also important for fine-tuning the innate immune response to infection and for the first step of adaptive immunity involving antigen-presenting cell (macrophages, dendritic cells)-related antigen presentation (Morris et al., 2013

According to these data, the authors proposed a model in which a decrease in PPAR expression in the rather chronic DM situation would impair expression of PPAR and Mfn2, thereby inducing mitochondrial fission which results in mitochondrial respiratory dysfunction, increased mitochondrial ROS generation and mitochondria-dependent apoptosis [114]