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tuberculosis through humoral immunity, with IgM titres correlating with reduced bacterial burden

Cephalalgia 26:816819

This mechanism ensures that, at low blood glucose levels, the liver does not compete with other organs, primarily the brain, for glucose

Virulent factor ESAT-6 drives macrophage differentiation toward the pro-inflammatory M1 phenotype and subsequently switches it to the anti-inflammatory M2 phenotype