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Interestingly, adoptive cell transfer with T cells lacking the Nox prevented ATII-induced increase in vascular ROS production and blood pressure in this animal model

Given that cuproptosis preferentially targets OXPHOS-dependent cells by disrupting TCA cycle enzymes and Fe-S cluster integrity, 167,213 it remains an open question whether GSH-dependent Fe-S cluster biogenesis similarly licenses susceptibility to cuproptosis