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In human brain, these neurons are thought to be particularly vulnerable due to their high level of pro-oxidant radical generation during decades of normal ageing

Herz 2007

To investigate how CI deficiency affects adipocyte biology, we generated mice lacking the core catalytic subunit of CI, Ndufs2 , specifically within adipocytes 29

Thus, controlling inflammation is a critical therapeutic strategy for preventing -cell loss and improving clinical outcomes and long-term prognosis in patients with T1DM

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