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at 0.667 mg/mL, 1 unit = 0.01 mL 6.7 mcg

A promising priority lies in developing dual-target nanotherapeutics capable of concurrently suppressing ferroptosis drivers (e.g., ACSL4) and amplifying immunoregulatory pathways (e.g., Treg activation), thereby enabling synergistic correction of both redox and immune dysfunction