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From a translational standpoint, the molecular complexity of HS highlights the urgent need for robust biomarkers of disease activity, progression, and therapeutic response

Future work is also needed to better understand how the BRD4/ASXL1/MLL3/4 epigenetic machinery and its associated transcriptional programs can be therapeutically targeted for cancer treatment

Research indicates that the DHODH pathway can protect cells from oxidative stress and mitochondrial damage through various mechanisms, including downregulating ROS levels, maintaining mitochondrial membrane potential, and inhibiting cell apoptosis (77, 78)

Effect of vitamin d3 on mitochondrial biogenesis in granulosa cells derived from polycystic ovary syndrome

Tang et al., 2013)