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for such mechanism full activation (opening) of SV/TPC1 channels, which is only observed at rather high cytosolic Ca 2+ , is not necessary so that the channel protein may operate in a non-canonical ion flux-independent way (Pottosin & Dobrovinskaya, Reference Pottosin and Dobrovinskaya2022)

Yesheng et al

Int Rev Neurobiol (2021) 156:162

In 3-KO mice, these reductions were absent, reinforcing the idea that 3-AR plays a necessary role in regulating the response of brown adipose tissue to these agents [2]