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3.4 Thyroid-specific oxidative stress and neuroinflammation Oxidative stress and neuroinflammation are key mechanisms linking thyroid dysfunction to neuronal injury

Lactase activity decreases after weaning

10.1016/j.ebiom.2015.10.018 22 BaiJ.BarandouziZ

Desmond, E

Therefore, decreased NCOA4 expression and impaired binding of NCOA4 to FTH1 diminish the FTH1 degradation by downregulating ferritinophagy, and decreased FPN1 expression inhibits iron output and release to other tissues, both of which effectively increase liver iron overload and, in turn, trigger oxidative stress and ferroptosis (82)