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Acylcarnitines that accumulate through disordered carnitine metabolism directly inhibit the surface activity of pulmonary surfactants and are considered risk factors for increased lung injury due to a second hit, such as respiratory tract infection, sepsis, or inhalation of toxins, in patients with dysregulated FAO [304, 305]

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LIN28B is required for LIP-induced metabolic reprogramming In the search for regulators of cell metabolism that use post-transcriptional mechanisms and are regulated by C/EBP, we re-analysed the transcriptome data

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