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As the deteriorating effect of inactivation of sensory nerves was significantly attenuated by co-treatment with exogenous CGRP, which is believed to account for the protective effects of sensory nerve excitation on gastric mucosa, it is reasonably to conclude that tryptophan protection against WRS-provoked gastric lesions could be attributed to their activation of sensory nerves and their release of CGRP protecting the mucosa (51, 52)

Furthermore, compared to the headfoot tissues, the differences of protein expression after O

siE1&2 vs siE1&2+Flag-Sororin **P = 0.0028, siE1&2 vs siE1&2+Flag-Sororin-12A **P = 0.0017, siE1&2 vs siE1&2+ Olaparib(+) ****P = 3.2485e-6

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Tracer experiments with labelled 13C-glutamine showed an enrichment of 13C carbons in glutathione