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Nevertheless, experimental data suggest that the altered intracellular redox status associated with hyperhomo-cysteinemia-induced oxidant stress is the major molecular mechanism responsible for the atherothrombogenicity of moderately elevated circulating homocysteine

10.1111/jcmm.12784 145 PernetE.SunS.SardenN.GonaS.NguyenA.KhanN.et al (2023)

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Evidence from these findings demonstrates that the disruption of these processes leads to the lungs inability to maintain homeostasis and recover from injury, resulting in a reduction in mucociliary clearance

Plasma soluble CD30 as a possible marker of adult T-cell leukemia in HTLV-1 carriers: a nested case-control study