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[12] During ketosis, the brain adapts to lower levels of available glucose by changing its fuel source to rely on ketone bodies for energy

Clin Microbiol Rev 1995

N., Parfrey, P

The involvement of miR-25 in the regulation of apoptotic marker genes via downregulation followed by recovery of response upon reconstitution, as well as the EGCG-mediated induction of apoptosis and interference with cell cycle progression (G2/M block), provide evidence that miR-25 may be a regulator of the apoptotic effects created by EGCG in these cells