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Therefore, HGF from neighboring cells in the liver and from extrahepatic organs may function as a trigger for liver regeneration by booth paracrine and endocrine mechanisms [44]

Because NO regulates vasodilation, endothelial survival, and angiogenic signaling, this balancing may support perfusion of injured cellular structures and facilitate endothelial activation and vessel remodeling during repair

Future research will need to clarify the extent to which these mechanisms are translatable into clinical practice and what their long-term implications may be for the human population

The results have been consistently promising across multiple tissue types and organ systems, though large-scale human clinical trials remain limited

Patients should avoid self-sourcing or using it without clinical guidance