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Rawji, K

This restores Wnt signaling defects in IECs, prevents hyponatremia, hypochloremia, and dehydration, and normalizes intestinal physiological functions in C

orthotopic models more accurately replicate TME interactions and metastasis but are technically challenging

1i) suggesting a compensatory mechanism for OPC loss, possibly involving bFGF (Supplementary Fig

Tissue biopsy results revealed enhanced collagen deposition in the AA-MNs group, and the proliferation marker Ki67 of keratin-forming cells exhibited elevated levels in MNs loaded with AA, demonstrating a higher potential for tissue regeneration and healing (Ryall et al., 2022a)