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Olesen, U

This phenotypic transitionfrom a protective, surveillant state to a pro-inflammatory and potentially detrimental phenotyperepresents a key mechanism underlying neuroinflammation and neurodegeneration in AD

Phase II randomized, placebo-controlled trial of green tea extract in patients with high-risk oral premalignant lesions

The lack of function of the enzymes of glutathione production in these conditions suggests that more efficient resolution of the effects of glutathione depletion may require the administration of the complete molecule as demonstrated in vitro in the HIV+ macrophage study [52]

But before diving into a protocol, its crucial to evaluate the potential risks, side effects, and unknowns especially for long-term use