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The nephrotoxic mechanisms of pCS primarily involve inducing oxidative stress through activation of nicotinamide adenine dinucleotide phosphate (NADPH) oxidase, triggering inflammatory responses, and promoting renal fibrosis (124)

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High vitamin D-binding protein concentration, low albumin, and mode of remission predict relapse in Crohns disease

Apigenin lead to increased zonulin 1 (ZO-1), claudin-1 and occludin expressions to restore the integrity of the intestinal barrier, regulating the microbial populations of Akkermansia, Turicibacter, Klebsiella, Romboutsia, etc., and its metabolites (SCFAs), thus attenuating DSS-induced colon injury [64]