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Tendon fibroblast studies have additionally documented BPC-157 upregulation of growth hormone receptor (GHR) expression, a receptor that signals canonically through the JAK2-STAT pathway [2]

Archived from the original on 4 March 2012

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However, studies using beta-3-adrenergic receptor knockout mice revealed a more complex picture: Long-term AOD-9604 treatment failed to produce weight loss in beta-3-AR knockout mice Acute administration still increased energy expenditure and fat oxidation in knockout mice This suggests beta-3-AR expression enhancement contributes to, but does not directly mediate, lipolytic effects The peptide appears to work through an unidentified receptor mechanism that subsequently influences beta-3-adrenergic signaling pathways