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Discussion Recent studies have begun to explore the roles of GSH in T cell activation and metabolic reprogramming (18, 19)

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This can trigger an endothelial-to-mesenchymal transition (94), where the loss of endothelial-specific markers and gaining of a mesenchymal or myofibroblastic phenotype contribute to fibrosis development due to the generation of activated myofibroblasts (95)