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the present review integrates these multi-axis regulatory pathways into a unified mechanistic framework, illustrating how mitochondrial damage-associated inflammatory signaling, mechanical stress-induced oxidative injury and metabolic reprogramming-driven lipid peroxidation collectively contribute to ferroptosis in tendon pathology (Fig

Identification of oxidant-sensitive proteins: TNF-alpha induces protein glutathiolation

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Santos-Neto et al., 2019), in the African meningitis belt, it is between 10 and 20% but reaches 80% during outbreaks (Tefera et al., 2020), while in Asian countries it is generally low (1.5-9.1%) (Serra et al., 2020)