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The postnatal NR1 knockout mutant mice, a model which produces early postnatal inhibition of NMDA-R activity in corticolimbic GABAergic interneurons, contributes to the onset of SCZ-like abnormalities (novelty-induced increase in locomotion, mating and nest-building difficulties, anxiety and anhedonia-like behaviors) in adult rodents

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Genome-wide association studies of metabolites in Finnish men identify disease-relevant loci

Moreover, alterations in the human gut-associated microbiome have been implicated in the pathogenesis of several chronic conditions ranging from atherosclerosis and thrombosis to hypertension and HF (Tang and Hazen, 2017) (Figure 1)

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