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UVC exposure predominantly activates lncRNAs that cooperate with p53 to regulate DNA damage response, apoptosis, and cell cycle arrest, vital for maintaining melanocyte genome integrity under severe UV stress 104

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This dysregulation is partially mediated by alterations in the NKCC1/KCC2 expression ratio, which governs intracellular chloride gradients and GABAergic polarity (see Section 'Disruption of Neurotransmitter Balance by Anesthetics'), as well as through direct interference with the functional integrity of GABA A Rs and NMDA receptors (Cabrera et al

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Calcifediol Treatment and Hospital Mortality Due to COVID-19: A Cohort Study