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The second key strategy was the overproduction of CgCS and CitS

In addition, oxidative glutamate toxicity is higher than the glutamate concentration required for NMDA receptor activation (Murphy et al., 1989)

88 In these experiments, we identified a window where drug metabolism and NAPQI formation were completed, but GSH could still effectively protect due to its scavenging of peroxynitrite, suggesting that this oxidant is critical for APAP-induced liver injury