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Deficiency of cysteine and GSH are associated with numerous etiologically unrelated diseases including skeletal muscle wasting and cancer 1

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During the progression of PF, various induction factors, such as cigarette smoke [25], bleomycin (BLM) [24], paraquat (PQ) [26], silicosis [27], and PM2.5 [28], have been shown to trigger the initiation of ferroptosis, leading to fibroblast-to-myofibroblast differentiation and pro-fibrotic factors release

Biologically significant markers on MAIT cells encompass the V7.2 TCR, co-stimulatory molecules (CD8 + / , CD4 + / , CD28), major activation markers (HLA-DR, CD69, CD39, CD38), the coinhibitory molecule LAG-3, and PD-1