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After 6 h of OGD, both exogenous and mutant TIGAR failed to rescue NADPH loss in cells, reproducing the PPP deficiency observed in ischemic mouse brains, but both wild-type and mutant TIGAR significantly reversed ROS burst in cells (Liu et al., 2022)

This represents one of the most critical intrinsic factors, rather than mobile genetic elements, associated with antibiotic resistance, usually resulting from their overexpression

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