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However, when the intensity of heat stress exceeds the cellular tolerance threshold, the cytoprotective effects of HSPs may be overwhelmed, permitting secondary inflammatory cascades (e.g., HMGB1/TLR4-mediated NF-B activation) to dominate, thereby exacerbating hepatocyte damage [16]

doi: 10.1089/jmf.2006.9.459 43 MoeinianMFarnaz Ghasemi-NiriSMozaffariSAbdollahiM

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8 Conclusion Compelling scientific evidence now establishes that environmental endocrine disruptors pose profound threats to male reproductive health through complex molecular mechanisms involving hormonal disruption, oxidative stress induction, mitochondrial dysfunction, and epigenetic modifications

Recent trends and challenges in interpretation of human biomonitoring data