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This review summarises the role and mechanisms of mitochondrial dysfunction in RA and discusses the potential and challenges of mitochondria as a novel therapeutic strategy for RA, highlighting the potential of mitochondria as a promising breakthrough point for the prevention and treatment of RA

10.1152/jappl.1993.75.6.2419 J

The tide began to turn in 1988 when research revealed that TNF- could trigger both apoptotic and necrotic cell death, suggesting that necrosis may not be a mere accident but a controlled process

Novel synthetic derivatives demonstrate increased half-life and reduced susceptibility to oxidation compared to natural EGCG