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Furthermore, our study suggested that at the acute stage of the disease, gut microbiota might exert a beneficial role in maintaining the homeostasis of BA and PUFA via an FGF19 signaling pathway and PUFA saturation respectively, although it seems that this negative feedback cannot totally counteract the accumulation of PUFAs and BAs

Thus, when DSBs happen, the NF-B complex is released to cause IFN- and IFN- production in regulating ATM [180, 181]

Brain-immune interactions and disease susceptibility

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Root responses to flooding