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Furthermore, treatment with Lumacaftor, a CFTR corrector facilitating the trafficking of misfolded F508del-CFTR protein, led to a reduction of superoxide levels in CFBE41o-cells, suggesting that while Lumacaftor primarily addresses CFTR protein processing, its therapeutic efficacy might also stem from mitigating oxidative damage and subsequent inflammatory responses (de Bari et al., 2018)

To preserve biological variance and ensure accurate correlation, raw data from all 15 individual biological replicates were directly paired with their corresponding transcriptomic profiles

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DRIP205 co-activator overexpression enhances PPARgamma-mediated differentiation of leukemia cells by CDDO

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