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Independent samples of the 40 molecules that met these criteria were re-purchased and counter-screened against WT (strain H99) and the acl1 mutant under screening conditions

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In both in vivo and in vitro experiments, promoting HSC ferroptosis through various pathways, such as inhibiting ACSL4 methylation [79] or sarcoma protein kinase activation [80], as well as regulating circDCBLD2-related mechanisms [81] and MafG/MYH9-LCN2 signaling pathway [82], can effectively alleviate liver fibrosis by reducing activated HSCs and collagen deposition