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Data availability about the pathophysiological processes involved in triggering NIHL has allowed researchers to use compounds, that could act as effective therapies, by targeting specific mechanisms such as the excess generation of free radicals and blood flow restriction to the cochlea

Molecular basis for the recognition of primary microRNAs by the Drosha-DGCR8 complex

Ammonium tetrathiomolybdate relieves oxidative stress in cisplatin-induced acute kidney injury via NRF2 signaling pathway

This seemingly simple mechanism has profound downstream consequences across virtually every tissue type, as actin dynamics are fundamental to cell migration, structural integrity, and signalling

Rather than forcing the system to do something unnatural, it simply provides a stronger version of the signal the body already uses