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Ferroptosis signaling activates PS exposure to promote axonal fusion It has been previously reported that phosphatidylserine (PS) externalization in PLM axon after injury serves as an important save me signal for the regrowing axon to recognize and connect 16

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ROS levels are increased in T cells with G6PD inhibition 98 , and glucose restriction in CD8 + T cells is sufficient to cause a decrease in the glutathione/glutathione disulfide ratio

SRs, particularly SR-A, have been implicated in neutrophil activation, including via mitogen-activated protein kinase (MAPK) signaling pathways, leading to increased production of proinflammatory cytokines (i.e., IL-6, TNF-) and NET formation (218)

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