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In an animal study involving Ang-II-induced hypertension, the researchers reported that in addition to the increase in blood pressure, the Ang-II-treated group also had increased expression of DNMT1 and DNMT3A, which act as an antioxidant defence systems to eliminate ROS (113)

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In therapeutic dosage, paracetamol is largely metabolised by phase II processes (glucuronidation and sulphation) to stable metabolites, but between 5% and 10% also undergoes P450 metabolism to the toxic N -acetyl p-benzoquinoneimine (NAPQI) metabolite (Nelson, 1990) (Figure 8.3)