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Propionate and acetate are additional substrates for the TCA cycle and ameliorate metabolic and intestinal diseases by activating intestinal gluconeogenesis (like microbiome-derived succinate 117 ) 118 and by enhancing innate immune responses via free fatty acid receptor 2 (FFAR2)-signaling 119

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MAK117, Sigma-Aldrich) was used to quantify glycerol release in extracellular media after sorafenib treatment

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