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High levels of PAGln are independently linked to a higher risk of coronary in-stent restenosis, which is linked to a poorer prognosis for CAD patients (128)

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In mice, deletion of REV3L resulted in impaired wound healing and excessive proliferation of the epidermis (69)

Our observation that intracellular NA concentrations did not increase following NNMT inhibition extends previous studies that showed NA concentrations were largely unaffected following NNMT knockdown with either adenovirus[39] or anti-sense oligonucleotides (ASOs).[12] It is possible that significant accumulation of NA in adipocytes (and other tissues) is prevented by rapid conversion of NA to NMN and subsequent NAD + synthesis or transport to the extracellular environment.[27] Importantly, NNMT inhibition potentiated the absolute intracellular concentrations of NAD + and SAM, without altering the levels of NA and SAH in the adipocytes