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Moreover, the significant implications of NRF2 hyperactivation, KEAP1 mutations, and BACH1 stabilization in cancer initiation, progression, and therapy resistance highlight the promising prospects of targeting the NRF2BACH1 signaling axis for therapeutic interventions in cancer management

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The accumulation of AGEs has been associated with several cardiovascular complications, including atrial fibrillation (AF) [41], congestive heart failure (CHF) [42], and coronary artery disease (CAD) [43] in individuals with T2DM

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Granulocyte-macrophage colony-stimulating factor (CSF) and macrophage CSF-dependent macrophage phenotypes display differences in cytokine profiles and transcription factor activities: implications for CSF blockade in inflammation