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Second, ferroptosis prevented by GPX4 might be one of the mechanisms that explain the relative immortal nature of cancer cells that contributed to poor prognosis in patients with GPX4-positive NSCLC in this study

It is tempting to speculate that the interaction between GABAergic and glutamatergic synapses could involve a bidirectional crosstalk that include signals from the excitatory synapse that regulate GABAergic synapse strength and persistence (Marsden et al., 2010)